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Excessive production of ROS disturbs mitochondrial function, damaging the mitochondrial membrane through lipid peroxidation (Shohami and Kohen, 2011)

These results implicate that targeting HDAC4 may become a promising approach for the treatment of epilepsy

Changes in DNA methylation add biological plausibility to the increasingly recognized contribution of environmental chemicals to disease burden [1] as DNA methylation is involved in regulating many cellular processes, including X-chromosome inactivation, genomic imprinting, chromosome stability, and gene transcription

10.7326/aitc202105180 82 McClureE

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